Yeah, the info in all the publications seems very repetitive... I was hoping to find some new info. I was wondering if you misted shipped eggs with Tylan etc. before you put them in the bator, and it absorbed into the egg if it would help cut down on the risk. It mentions it briefly in a publication, but not amount of, how to or how well.......
Edited by chickenzoo - 12/5/09 at 2:06pm
I keep seeing more and more people mention about a cough, or runny nose, sneezing ,a bubbly or swollen mucus y eye in their birds, which all point in the way of MG or Corzya, or both, which seems to be happening more and more......
This is what was sent to me when I inquired about it in our state....
Synonyms: MG, chronic respiratory disease (CRD), infectious sinusitis, mycoplasmosis
Species affected: chickens, turkeys, pigeons, ducks, peafowl and passerine birds.
Clinical signs: Clinical symptoms vary slightly between species. Infected adult chickens may show no outward signs if infection is uncomplicated. However, sticky, serous exudate from nostrils, foamy exudate in eyes, and swollen sinuses can occur, especially in broilers. The air sacs may become infected. Infected birds can develop respiratory rales and sneeze. Affected birds are often stunted and unthrifty (see Table 1 <http://edis.ifas.ufl.edu/ps044#TABLE_1> ).
There are two forms of this disease in the turkey. With the "upper form" the birds have watery eyes and nostrils, the infraorbitals (just below the eye) become swollen, and the exudate becomes caseous and firm. The birds have respiratory rales and show unthriftiness.
With the "lower form", infected turkeys develop airsacculitis. As with chickens, birds can show no outward signs if the infection is uncomplicated. Thus, the condition may go unnoticed until the birds are slaughtered and the typical legions are seen. Birds with airsacculitis are condemned.
MG in chicken embryos can cause dwarfing, airsacculitis, and death.
Transmission: MG can be spread to offspring through the egg. Most commercial breeding flocks, however, are MG-free. Introduction of infected replacement birds can introduce the disease to MG-negative flocks. MG can also be spread by using MG-contaminated equipment.
Treatment : Outbreaks of MG can be controlled with the use of antibiotics. Erythromycin, tylosin, spectinomycin, and lincomycin all exhibit anti-mycoplasma activity and have given good results. Administration of most of these antibiotics can be by feed, water or injection. These are effective in reducing clinical disease. However, birds remain carriers for life.
Prevention: Eradication is the best control of mycoplasma disease. The National Poultry Improvement Plan monitors all participating chicken and turkey breeder flocks.
Synonyms: MS, infectious synovitis, synovitis, silent air sac
Species affected: chickens and turkeys.
Clinical signs: Birds infected with the synovitis form show lameness, followed by lethargy, reluctance to move, swollen joints, stilted gait, loss of weight, and formation of breast blisters. Birds infected with the respiratory form exhibit respiratory distress. Greenish diarrhea is common in dying birds (see Table 1 <http://edis.ifas.ufl.edu/ps044#TABLE_1> ). Clinically, the disease in indistinguishable from MG.
Transmission: MS is transmitted from infected breeder to progeny via the egg. Within a flock, MS is spread by direct contact with infected birds as well as through airborne particles over short distances.
Treatment: Recovery is slow for both respiratory and synovitis forms. Several antibiotics are variably effective. The most effective are tylosin, erthromycin, spectinomycin, lincomycin, and chlorotectracycline. These antibiotics can be given by injection while some can be administered in the feed or drinking water. These treatments are most effective when the antibiotics are injected.
Prevention: Eradication is the best and only sure control. Do not use breeder replacements from flocks that have had MS. The National Poultry Improvement Plan monitors for MS.