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Discussion in 'Emergencies / Diseases / Injuries and Cures' started by ravenking, Feb 12, 2017.
my hen started sneezing what to do and what could be wrong i use hay as bedding
Any other symptoms?
not that i saw nose might be a little runny but not alot i took out the straw to see if it helpd any
Is the sneezing once in awhile or every few minutes? Do you see any nasal secretions or dirt around the nostrils, any watery eyes or drainage, or rattly breathing. Dust, mold, and poor air circulation could be problems, but if you are seeing frquent sneezing, it may be a respiratory disease such as infectious bronchitis or others.
i think shes sick whats good medicine to give her i seperated her already
For a respiratory disease, Tylan50 injectable is about all you will find in feed stores without a prescription. Dosage is 1 ml twice a day for 5 days for a five pound chicken. Get some syringes and needles to withdraw the medicine. You can give it orally or as a shot in the breast muscle. It can treat mycoplasma and some bacterial diseases, but if she has bronchitis or another virus, it might not help that much. Viruses will have to run their course over several weeks. An antibiotic like Tylan could possibly help to prevent secondary infections. Hay and straw can become dusty or wet and moldy, so watch out for that since it can cause sneezing and breathing problems. Provide good overhead air circulation in the coop. Let us know how she is doing tomorrow.
Here are some of the most common diseases in a link from University of FL
Synonyms: IB, bronchitis, cold
Species affected: Infectious bronchitis is a disease of chickens only. A similar disease occurs in bobwhite quail (quail bronchitis), but it is caused by a different virus.
Clinical signs: The severity of infectious bronchitis infection is influenced by the age and immune status of the flock, by environmental conditions, and by the presence of other diseases. Feed and water consumption declines. Affected chickens will be chirping, with a watery discharge from the eyes and nostrils, and labored breathing with some gasping in young chickens. Breathing noises are more noticeable at night while the birds rest. Egg production drops dramatically. Production will recover in 5 or 6 weeks, but at a lower rate. The infectious bronchitis virus infects many tissues of the body, including the reproductive tract (see Table 1). Eggshells become rough and the egg white becomes watery. (See publication PS-24, Egg Quality, for other causes of poor egg quality.)
Transmission: Infectious bronchitis is a very contagious poultry disease. It is spread by air, feed bags, infected dead birds, infected houses, and rodents. The virus can be egg-transmitted, however, affected embryos usually will not hatch.
Treatment: There is no specific treatment for infectious bronchitis. Antibiotics for 3–5 days may aid in combating secondary bacterial infections. Raise the room temperature 5°F for brooding-age chickens until symptoms subside. Baby chicks can be encouraged to eat by using a warm, moist mash.
Prevention: Establish and enforce a biosecurity program. Vaccinations are available.
Synonyms: MG, chronic respiratory disease (CRD), infectious sinusitis, mycoplasmosis
Species affected: chickens, turkeys, pigeons, ducks, peafowl, and passerine birds.
Clinical signs: Clinical symptoms vary slightly between species. Infected adult chickens may show no outward signs if infection is uncomplicated. However, sticky, serous exudate from nostrils, foamy exudate in eyes, and swollen sinuses can occur, especially in broilers. The air sacs may become infected. Infected birds can develop respiratory rales and sneeze. Affected birds are often stunted and unthrifty (see Table 1).
There are two forms of this disease in the turkey. With the "upper form" the birds have watery eyes and nostrils, the infraorbitals (just below the eye) become swollen, and the exudate becomes caseous and firm. The birds have respiratory rales and show unthriftiness.
With the "lower form", infected turkeys develop airsacculitis. As with chickens, birds can show no outward signs if the infection is uncomplicated. Thus, the condition may go unnoticed until the birds are slaughtered and the typical legions are seen. Birds with airsacculitis are condemned.
MG in chicken embryos can cause dwarfing, airsacculitis, and death.
Transmission: MG can be spread to offspring through the egg. Most commercial breeding flocks, however, are MG-free. Introduction of infected replacement birds can introduce the disease to MG-negative flocks. MG can also be spread by using MG-contaminated equipment.
Treatment: Outbreaks of MG can be controlled with the use of antibiotics. Erythromycin, tylosin, spectinomycin, and lincomycin all exhibit anti-mycoplasma activity and have given good results. Administration of most of these antibiotics can be by feed, water or injection. These are effective in reducing clinical disease. However, birds remain carriers for life.
Prevention: Eradication is the best control of mycoplasma disease. The National Poultry Improvement Plan monitors all participating chicken and turkey breeder flocks.
Synonyms: roup, cold, coryza
Species affected: chickens, pheasants, and guinea fowl. Common in game chicken flocks.
Clinical signs: Swelling around the face, foul smelling, thick, sticky discharge from the nostrils and eyes, labored breathing, and rales (rattles—an abnormal breathing sound) are common clinical signs. The eyelids are irritated and may stick together. The birds may have diarrhea and growing birds may become stunted (see Table 1).
Mortality from coryza is usually low, but infections can decrease egg production and increase the incidence and/or severity of other diseases. Mortality can be as high as 50 percent, but is usually no more than 20 percent. The clinical disease can last from a few days to 2–3 months, depending on the virulence of the pathogen and the existence of other infections such as mycoplasmosis.
Transmission: Coryza is primarily transmitted by direct bird-to-bird contact. This can be from infected birds brought into the flock as well as from birds which recover from the disease which remain carriers of the organism and may shed intermittently throughout their lives. Birds risk exposure at poultry shows, bird swaps, and live-bird sales. Inapparent infected adult birds added into a flock are a common source for outbreaks. Within a flock, inhalation of airborne respiratory droplets, and contamination of feed and/or water are common modes of spread.
Treatment: Water soluble antibiotics or antibacterials can be used. Sulfadimethoxine (Albon, Di-Methox) is the preferred treatment. If it is not available, or not effective, sulfamethazine (Sulfa-Max, SulfaSure), erythromycin (gallimycin), or tetracycline (Aureomycin) can be used as alternative treatments. Sulfa drugs are not FDA approved for pullets older than 14 weeks of age or for commercial layer hens. While antibiotics can be effective in reducing clinical disease, they do not eliminate carrier birds.
Prevention: Good management and sanitation are the best ways to avoid infectious coryza. Most outbreaks occur as a result of mixing flocks. All replacement birds on "coryza-endemic" farms should be vaccinated. The vaccine (Coryza-Vac) is administered subcutaneously (under the skin) on the back of the neck. Each chicken should be vaccinated four times, starting at 5 weeks of age with at least 4 weeks between injections. Vaccinate again at 10 months of age and twice yearly thereafter.
Synonyms: LT, ILT, trach, laryngo
Species affected: Chickens and pheasants are affected by LT. Chickens 14 weeks and older are more susceptible than young chickens. Most LT outbreaks occur in mature hens. In recent years, LT has also caused significant respiratory problems in broilers greater than 3 weeks of age, especially during the cooler seasons of the year. This is believed to be due to unwanted spread of LT vaccines between poultry flocks.
Clinical signs: The clinical sign usually first noticed is watery eyes. Affected birds remain quiet because breathing is difficult. Coughing, sneezing, and shaking of the head to dislodge exudate plugs in the windpipe follow. Birds extend their head and neck to facilitate breathing (commonly referred to as "pump handle respiration"). Inhalation produces a wheezing and gurgling sound. Blood-tinged exudates and serum clots are expelled from the trachea of affected birds. Many birds die from asphyxiation due to a blockage of the trachea when the tracheal plug is freed (see Table 1).
Transmission: LT is spread by the respiratory route. LT is also spread from flock to flock by contaminated clothing, shoes, tires, etc. Birds that recover should be considered carriers for life. LT may be harbored in speciality poultry such as exhibition birds and game fowl.
Treatment: Incinerate dead birds, administer antibiotics to control secondary infection, and vaccinate the flock. Mass vaccination by spray or drinking water method is not recommended for large commercial or caged flocks. Individual bird administration by the eye-drop route is suggested. Follow manufacturers instructions. In small poultry flocks, use a swab to remove plug from gasping birds, and vaccinate by eye-drop method.
Prevention: Vaccinate replacement birds for outbreak farms. Vaccination for LT is not as successful as for other disease, but is an excellent preventive measure for use in outbreaks and in epidemic areas. Refer to the publication PS-36 (Vaccination of Small Poultry Flocks) for more information on LT vaccinations.