Quote:
I've posted the following info before, but I don't remember whether it was in this thread or somewhere else. Here it is again!
syndactyly = fused toes
ptilopody = feathered feet and toes
brachydactyly = shortened toes
"Syndactyly is a condition in which the third and fourth toes are
fused, usually by continuous webbing between the toes (Figure 6.6).
The extent of webbing varies distally and between toes. In extreme
cases the toes are tightly tied together, and the nail of the fourth
toe may be bent out of its median position making walking difficult.
The condition is usually expressed in both feet. Syndactyly has been
associated with both ptilopody and brachydactyly by several workers
(Davenport, 1909; Danforth, 1919a, 1919b; Jaap, 1939). They proposed
that the same gene was responsible for all three of these conditions
and that modifying genes and genetic background determine the
phenotype of each individual. Davenport's (1909) data showed
syndactyly to be dominant. This would be expected if one of the
dominant shank feathering genes were responsible for it. Danforth
(1929) suggested that both of the dominant shank feathering genes
were able to produce syndactyly. A relationship between these two
traits was further strengthened with the appearance of the single
gene multitrait mutant psp reported by McGibbon and Shackelford (192)
and Smyth (1981) in which syndactyly and ptilopody were shown to be
caused by the same recessive gene.
There probably is more than one genetic type of syndactyly. Two
studies have been made of this trait in clean-legged stock, where
ptilopody genes could not be involved. One study used White Plymouth
Rocks (Warren, 1950) and the other one involved junglefowl stock
(Hollander and Brumbaugh, 1969). In both cases, results from crosses
were difficult to interpret. Warren (1950) found that the normal
parent had a major influence on F1 progeny; he got only 1.8 percent
syndactylism in one group of F1 crosses but 41 percent in another,
syndactyl x syndactyl crosses produced 62 percent affected progeny,
and there were only 11.9 percent affected in the F2 generation. The
comparable results from Hollander and Brumbaugh (1969) were zero, 40
and one percent affected birds. Although Warren (1950) had somewhat
more affected individuals, these two groups may well have been
working with the same genetic complex. Hollander and Brumbaugh (1969)
proposed complementary gene action of a dominant and a recessive
factor along with variable penetrance and expressivity. Warren (1950)
did not suggest a mode of inheritance, but said that there was no
evidence of sex-linkage."
pp. 201-202, Poultry Breeding and Genetics -- Roy D. Crawford,
Elsevier, 1990.
Thanks Amazondoc