I am not sure why there is such an antipathy to the Phoenix introduction, because, as you say, the feathering on the saddle indicates individuals carry Gt as well as the Gt^Sd alleles.Gt also affects the hackle, breast feathering snd length of the main wing feathers— but does not DIRECTLY effect the saddle!, which Reeder says appears to be controlled by a separate factor he named Gt^ Sd that appears to operate as an autosomal dominant.
When looking at saddle lengthening, Gt^Sd operating exactly the same way as the Gt gene, (with the homozygous showing more lengthening than the heterozygous.) To give an idea: normal saddle feathers are about 4 inches long. If you have the homozygous saddle gene, by itself, without the non-molting factor— you can expect up to 12 inches of saddle hackle to be expressed; with the heterozygous and again, non molting factors NOT present, you are looking at 6inches, approximately in length. And that’s valuable to know about these saddle feathers, how long they can be before you even start looking at adding the non molting gene. What is more important, is that this tells you Gt^Sd can be expressed EVEN IN THE ABSENCE of the Gt gene. That is what the Genetic Hackle Fowl breeders are more focused on — the Gt^Sd saddle hackle lengthening gene, not so much the tail. You have seen, I’m sure, photos of them holding birds with tremendously long saddle hackles, but a rather small tail. Dr. Tom Whiting is focusing on extreme length on the saddle hackle AND the neck hackle, for example.
It may be that the webless trait is not as fully expressed in Phoenix — Whiting himself said when he overhauled his Genetic Hackle Fowl Program, he introduced no less than 50 purebred show type Silkies to overwhelm the genetics of his flock with the webless factor.
It might be Phoenix have more of a tendency to have twist of the rachis( stem) on a vise for fly tying, though this is merely speculation.
I would suspect that some Phoenix may not carry the mt ( non molting) gene as heavily as the Genetic Hackle Breeders would like. It’s a quantitative trait, it’s difficult to describe, and called an autosomal recessive, but it’s not straightforward. It’s two major effects are causing blood feathers to stay in what’s called “perpetual growth” which actually just means a Very Long Time.
Secondly, that mt alleged autosomal recessive allele delays molting. I say alleged because Mr. Reeder is not at all convinced that the two different effects are the result of one gene—he has seen it occur separately, which raises doubts.
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