Brilliant, thanks Tim.
When you write partridge you refer to eb/eb? The use of the word partridge is a bit confusing to me. In UK eb/eb, Pg/Pg, s+/s+ Orps & Wyandottes are called partridge but with Welsumers the wild type is called partridge.
Few things I'd like to discuss; hope you don't mind...Comparing notes, especially on genes which don't seem to have been investigated can be very useful.
Interesting what you're saying about the delawares & the undocumented autosomal red inhibitor. Have you experienced the same or similar gene in any other breeds?
Thank you for the description of autosomal red. I haven't done much with RIR except nicking reddening genes from the very dark exhibition types. Embarrassingly, while I have "Genetics of the Fowl" I haven't read it all because quite a bit is out of date. That description of Autosomal Red makes more sense than any description I've heard. I've had it described as the gene which makes a gold duckwing gold.....but Clive Carefoot's description of gold duckwing is the one which works. I've had it described as the phaeomelanin which causes the salmon colour in females of wheaten & wild type & but that seems, to me, to be an effect of their allele at the e-locus.
In work with each of Orps & RIR both separately & together, I have experienced a few autosomal phaeomelanin producing genes, which seem to be acknowledged, but are probably not particularly important. But they can be useful. Apart from autosomal red in the RIR have you encountered other gold or reddening genes?
What do you know about the gene Li? It is mentioned in Poultry Breeding & Genetics under eumelanin restrictors but not it is not described. Apart from causing light down, from what parts does it restrict eumelanin? I ask because I'm wondering whether it might be the gene I observed in the German bred Red Orps.
The main line of buff orps I used, in UK, were ey & did not carry dominant white. We did have a few from another line which did carry dominant white.
Things seem to be done in such a different way over here. Top exhibition breeders in UK don't usually sell eggs so one sees what one is purchasing, but then Britain isn't very big. (Just great VVVBG) So, with top quality, stock, one tends to get what one expects. I purchased some speckled sussex eggs from a private breeder not long after I arrived in US, they appeared to be wild type at hatch; they were a mess. Is there a usual way of obtaining stock that is what it is supposed to be, at least genetically, without travelling thousands of miles to view the parent stock?
Sorry for all the questions. Hope you don't mind.
Hwyl Fawr
When you write partridge you refer to eb/eb? The use of the word partridge is a bit confusing to me. In UK eb/eb, Pg/Pg, s+/s+ Orps & Wyandottes are called partridge but with Welsumers the wild type is called partridge.
Few things I'd like to discuss; hope you don't mind...Comparing notes, especially on genes which don't seem to have been investigated can be very useful.
Interesting what you're saying about the delawares & the undocumented autosomal red inhibitor. Have you experienced the same or similar gene in any other breeds?
Thank you for the description of autosomal red. I haven't done much with RIR except nicking reddening genes from the very dark exhibition types. Embarrassingly, while I have "Genetics of the Fowl" I haven't read it all because quite a bit is out of date. That description of Autosomal Red makes more sense than any description I've heard. I've had it described as the gene which makes a gold duckwing gold.....but Clive Carefoot's description of gold duckwing is the one which works. I've had it described as the phaeomelanin which causes the salmon colour in females of wheaten & wild type & but that seems, to me, to be an effect of their allele at the e-locus.
In work with each of Orps & RIR both separately & together, I have experienced a few autosomal phaeomelanin producing genes, which seem to be acknowledged, but are probably not particularly important. But they can be useful. Apart from autosomal red in the RIR have you encountered other gold or reddening genes?
What do you know about the gene Li? It is mentioned in Poultry Breeding & Genetics under eumelanin restrictors but not it is not described. Apart from causing light down, from what parts does it restrict eumelanin? I ask because I'm wondering whether it might be the gene I observed in the German bred Red Orps.
The main line of buff orps I used, in UK, were ey & did not carry dominant white. We did have a few from another line which did carry dominant white.
Things seem to be done in such a different way over here. Top exhibition breeders in UK don't usually sell eggs so one sees what one is purchasing, but then Britain isn't very big. (Just great VVVBG) So, with top quality, stock, one tends to get what one expects. I purchased some speckled sussex eggs from a private breeder not long after I arrived in US, they appeared to be wild type at hatch; they were a mess. Is there a usual way of obtaining stock that is what it is supposed to be, at least genetically, without travelling thousands of miles to view the parent stock?
Sorry for all the questions. Hope you don't mind.
Hwyl Fawr