thank you. the sheep started having seizures randomly but no changes in diet or routine

male or female, age, pregnant, There are very many causes many can be contaminated ground
White muscle disease
The cause of white muscle disease (muscular dystrophy) is a lack of selenium or vitamin E or both. In Minnesota, a lack of vitamin E is more likely to be the cause. Signs are lambs born dead or weak, or lambs that are unable to rise or walk or that do so stiffly. It may affect six- to eight-week-old lambs. Very often the fastest gaining lambs are affected.

To prevent white muscle disease, feed salt containing 90 ppm selenium, feed salt fortified with 100,000 I.U. vitamin E per 100 pounds salt, or inject young lambs with selenium and vitamin E on day 1 and day 10.

Neurological diseases
  • Bacterial meningitis: The lamb can't stand and its rear quarter is weak. The brain is infected. Antibiotics may help but the prognosis is guarded.
  • Polioencephalomalacia: The disease occurs in feedlot lambs on high grain rations and is caused by a bacteria in the rumen that deprives the lamb of thiamine. In contrast to enterotoxemia, the lamb lives one to three days. Infected lambs flex back their heads and peddle their legs. Thiamine injection may produce a dramatic response if lambs are treated early.
  • Listeriosis: An aerobic bacteria, listeria, which frequently occurs in silage, causes this disease. The sheep circle around. Antibiotic treatment may save a small percentage of infected sheep. Feeding high quality, mold-free silage will not cause the disease.
  • Rabies: Skunks transmit rabies to sheep.
  • Tetanus: Tetanus is caused by Clostridium tetani, which persists in the soil of most farms. Next to horses, sheep are the most susceptible farm animal. The bacteria are anaerobic, so wounds in which air contact is limited are most susceptible to tetanus. Docking and castrating with rubber rings increase the incidence of infection. Disinfecting docking and castrating wounds will minimize it. Infected sheep become stiff, move with a straddled gait, and usually die. Vaccinating with tetanus toxoid and anti-toxin prior to docking is effective.

Enterotoxemia
Enterotoxemia can kill sheep of all ages but usually kills only those that consume high levels of carbohydrates. Feedlot mortality for vaccinated lambs is .5%; for unvaccinated lambs it is 5 to 10%. Vaccination costs about 15 cents per lamb.

Clostridium perfringens type D, one type of bacteria that causes enterotoxemia, is most prevalent in feedlot or in creep-fed lambs. Symptoms are sudden death, occasional pushing and staggering, and apparent blindness.

For an outbreak, vaccinate with type D toxoid on day 1 and again 12 to 14 days later, de-worm, and reduce grain until the vaccine takes effect.

C. perfringens type C causes a type of enterotoxemia that usually is accompanied by bloody scours. Mortality may be high. It usually occurs among fast gaining lambs during the first three weeks. Ewes vaccinated three-four weeks pre-lambing provide antibody protection in their milk. Normally, vaccine won't "take" on young (3– to 10–day) lambs that are nursing.

Acidosis: High grain intake lowers the rumen pH from about 7.2 to 5.2 to 5.8 causing lambs to discontinue eating, pain and if sufficiently severe, death. Purging with mineral oil or a bicarbonate drench is effective.
 

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