Crop not filling up?

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Although a long shot I had some birds get wet pox and the symptoms you’re describing is what these birds had. They had voracious appetites, never filled their crops and wasted away. I ended up making my own treatment since the urban vets here in a big city wouldnt treat them. My treatment did work and they eventually got better.

I just saw she passed away. Sorry for your loss.
 
bhaugh said:
Although a long shot I had some birds get wet pox and the symptoms you’re describing is what these birds had. They had voracious appetites, never filled their crops and wasted away. I ended up making my own treatment since the urban vets here in a big city wouldnt treat them. My treatment did work and they eventually got better.

I just saw she passed away. Sorry for your loss.
Click to expand...
Thank you. I’ll be interested in seeing the results, and really hope it’s not contagious. If it happens to be wet pox, I’ll surely bend your ear.
 
@Eggcessive @azygous @Wyorp Rock @casportpony

Dropped off Ethyl with the examiner this morning and was told we would get the report in 1-3 weeks. About 20 minutes later I received a call from the doctor. He had a very thick accent and was hard to understand, but what we did understand was ovarian and/or oviduct tumor/cancer with water in the belly (ascites). I specifically asked about Marek’s and worms, and he said negative for both. He also said I should have the report by the end of the week.

I started searching for more info about this, and found this to be a fairly common occurrence (I think up to 35%?), but in hens. I couldn’t find anything that spoke to this in pullets (she was almost 9 months old). I’m also not even positive she was laying.

I couldn’t find info on breed specificity related to this illness, either. I know I’ll have more info when the report comes in, but the doctor used a term that I can’t remember at this time, but it meant that it was the predominant cause of her death.

Do you all have info as it relates to pullets getting this disease? I’m thankful it wasn’t anything contagious, but really very curious on possible likelihood of it being prevalent in pullets, as well.

Thank you 🙏
 
These reproductive cancers can happen at most any age, but much more often in older hens. What does promote such a cancer in a young hen is carrying the lymphoid leucosis virus. It can cause tumors in chickens as young as just a few months. But if the necropsy did not find an enlarged liver and tumors on other organs, then it's less likely to have been caused by the leucosis virus.

So, the key would be an enlarged liver and tumors on other organs. Absent those markers, this cancer may have been from an unfortunate genetic predisposition.
 
azygous said:
These reproductive cancers can happen at most any age, but much more often in older hens. What does promote such a cancer in a young hen is carrying the lymphoid leucosis virus. It can cause tumors in chickens as young as just a few months. But if the necropsy did not find an enlarged liver and tumors on other organs, then it's less likely to have been caused by the leucosis virus.

So, the key would be an enlarged liver and tumors on other organs. Absent those markers, this cancer may have been from an unfortunate genetic predisposition.
Click to expand...
I will read up on that virus now. I wish I would have done the professional necropsy on Betty White - they were sisters, so the genetic component could have been telling.
 
Here is the body of the report. They put the wrong name - should have been Ethyl. I had to copy and paste because I couldn’t edit the report on my phone to delete personal info.


Species:
Breed:
23-12540
Betty
Gallus gallus Chicken, Domestic
Received:
04/26/23 Reported:
04/27/23 Final Report
A 9-month-old female chicken identified as "Betty" was submitted for necropsy examination. The animal was in fair body condition with prominent keel bone. The body weight was 2.4 lbs. The lungs were moderately congested. The coelomic cavity was markedly distended and contained large amounts of yellowish fluid (ascites). The ovary was enlarged by an approximately 6.5 cm in diameter multinodular, firm, white masses (cauliflower-like growths). Approximately 90 - 95 %of the ovarian tissue was replaced by these masses. Many of the nodules coalesced.The wall of the duodenum was thickened and adhered together and large portion of the pancreas in the duodenal loop was replaced by a similar, tan to white firm mass (transcoelomic implants). No other significant gross lesions were observed.

FINAL COMMENTS
The gross necropsy lesions are compatible with ovarian adenocarcinoma with transcoelomic implants and secondary ascites. nI this chicken, there was no evidence of an infectious disease that could be contagious to other chickens. No further laboratory work was
performed since there were no lesions found that would indicate that such testing would be helpful and the cause of death was determined.
CASE DIAGNOSIS
ADENOCARCINOMA- OVARIAN
ASCITES
 

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