Got the final neocropsy results in today. However, this is not going to be the last of it. Still waiting on toxicology on the feed tests, and viral tests for calcivirus, and whatever else could be causing it. The original is seven pages long. Since I don't want to type the whole thing, I am just going to post the basic findings. I think, by doing research on the net, that one of the findings means a lung, or rare lung, or heart infection/damage. Another means they could have had a viral type of e coli. Someone on the meat rabbitslist said that heart damage can be caused by bad feed. So we will see. Laboratory findingsfrom page 2) Fecal examination: few coccidial oocysts Respiratory culture: Few morganella morganii, rare enterococcus faecalis Liver: Rare oligella areolytica Page 3: Rabbit B: (rabbit B was the one with the broken back from the first neocropsy report) Heart: severe diffuse mycocardial loss and fibrosis Lung: Moderate diffuse serofribrinous interstitial pneomonia Kidney: Focal pygranuloma spinal cord: Moderate locally extensive vacuolar myelopathy Rabbit A: Heart: focal mild mycocardial loos and fibrosis Final diagnosis Rabbit B: Hepetatic necrosis(cause undetermined) Bacterial septecemia(suspected) Severe mycrocardial degeneration Vertebral fracture with subsequent spinal cord degeneration Rabbit A mild mycrocardial degeneration The most significant changes are within the liver. Possible differentials at this time include viral infection (such as by calcivirus) and acute aflatoxicosis. The changes within the lungs in addition to the pygranulomas within the liver and kidney suggest underlying bacteria septecemia: however, the bacteria cultured are most consistant with postmortem contaminants or opportunistic overgrowth rather that the the inciting cause. This may be due to antibiotic treatment prior to death. (dummy me gave them an accidental dose of terramycin). A clear cause of the mycocardial degeneration, seen in both rabbits, or its relation to the other lesions observed was not found. Vitamin E/selenium deficiency in rabbits is not clealry defined. Also goes on to say that virology tests are pending. So are feed tests. Page 5 talks about the bacteria culture in the lungs, and the antibiotics susceptible to it. page 6-7 says they found Escherichia coli in the intestine culture. No growth of samonella. Goes on to talk about the drugs suseptible to it. So according to that there may be a vriral test on its way, am I correct? I still doubt its the calcivirus. You'd think I'd still be seeing problems with them by now. I haven't had another death since they were dosed with oxytetracycline, and put on purina rabbit chow. Hopefully that right there has cleared up a lot that could be causing our animals to die. Thoughts anyone? So far I have got no response from the ARBA on the situation yet. However, one of the rabbit and cavy health commitee members (who is also a rabbit vet) saw my posts on the email lists and has been in contact with me over it. So through that person I assume they are aware of what is going on. The person who was supposed to send theirs for a neocropsy at Perdue(SP?), never followed through with it. They wanted to treat their herd with albon. I think I finally convinced them today that they need to forget about the albon, and get the dead bunnies tested. Their vet is sending one in a couple of days. I think the mention of the calcivirus, and the e coli strain may have changed their mind.