This is an extract from the Merck Veterinary Manual. I read a bunch of the scientific papers but actually this isn't a bad summary of what I read. Basically the issue seems to be in the liver making fat to produce adequate yolks for the eggs. So you need to maintain a good energy level for the chicken (they are not advocating getting the birds to be overweight) and to substitue carbohydrate with fat.Don't you think a thread on rescued battery hens is the right place for a discussion on the relative roles of different factors to explain their abnormally short lifespans?
So I've grouped responses by science, then environment, then examples, then diet.
Is this linked with genes? If so, surely there's research on how to inactive those genes in the seemingly endless search for (human) longevity? And if it's not linked to genes, what else more obvious than diet? These chickens demonstrate enhanced performance, and, to compare it with our enhanced performers (sportspeople), the chickens are not getting it through exercise, so that only really leaves diet, doesn't it?
'They are bred' implies genes again. Can you give me something heavy to read on this?
Very important point. The average masks a range of values.
Also a very important point. I am not at all suggesting genes don't matter. But I do think they are a useful distraction for the feed industry, because while we're hand-waving in the direction of genetics, about which we can do nothing of course, they can keep their heads down and try to stay out of the argument. A lot of human ill-health is attributed to diet, especially highly processed food (as your post so clearly showed); chicken ill-health seems to be chaotically attributed to anything they eat except commercial (i.e. highly-processed) feed, as Iluveggers pointed out so well.
Quantity and quality of exercise is another very good, and far too often overlooked, point.
Indeed. We make do with what we must!
Thanks for this concrete example Molpet. The other thing to consider with high production breeds is that they are very highly inbred. That in itself can lead to all sorts of problems, as very rare breeds and exhibition strains of pure breeds often show.
That's very interesting; the instinct to forage is not just still there, but it's strong.
That would explain the number of times I've seen the advice to give extra vit B! Thanks. Here it is automatically added to breakfast cereal, to make sure kids get enough whatever the nutritional nous of their parents, so it's easy and cheap to add to whatever we give as chicken feed if we feel supplementation is necessary.
I wish this were better known. There's a lot of rubbish said about fat per se.
***
Experimentally, most attempts to prevent or treat the condition have been made through dietary modification. Substituting carbohydrate with supplemental fat, while not increasing the energy content of the diet, seems to be beneficial. Presumably such modification means the liver needs to synthesize less fat for yolk. Replacement of corn with other cereals, such as wheat and barley, is often beneficial. However, this substitution may reduce the dietary energy level or necessitate using additional fat to maintain isoenergetic conditions, two factors known to influence FLHS. A wide energy

Various byproduct feeds such as distiller’s grains, fish meal, and alfalfa meal reduce the incidence of FLHS. Supplementation with selenium also has been shown to reduce FLHS, but the mode of action is unclear. When a farm has a history of FLHS, the diet should contain at least 0.3 ppm selenium, ideally as organic selenium, and up to 100 IU vitamin E/kg diet, with appropriate levels of an antioxidant such as ethoxyquin. There are reports of layers having greater incidence of fatty liver when fed chelated trace minerals versus conventional inorganic minerals. However, the relation between the increased organic minerals in layer diets and the incidence of FLHS is unknown. These various additives collectively help to limit the occurrence of tissue rancidity and thus hemorrhage of the excess fat in the liver.
https://www.merckvetmanual.com/poul...c syndrome is,and fat engorgement at necropsy.
***