update-Rudy's gone...Anyone here ever given their chicken glucosamine?

[TJ]

Hello, I have a Golden Campine rooster, he's about 6 mos. old and he has problems with his legs. His legs are swollen and in some areas they are the size of half dollars... I took him to the vet and he told me it was a bone/joint issue and that I should try mixing Clovite with the poultry feed and other than that there was really nothing he could do. Its been a couple of weeks with the Clovite and I haven't seen any improvement. His legs are so swollen that he sits most of the time and he manages to walk but it is more like a stumble...

I had heard from a friend recently about the wonders of glucosamine in humans and animals alike for bone, arthritis, joint, etc...issues. So this morning I made some oatmeal, mixed in a lil' cracked corn and crushed a half pill of glucosamine and mixed it all together. I just gave it to my lil' rooster and he ate the plate clean...I was thinking about doing this every morning and see if maybe this will help him. Has anyone here ever done this? Has anyone here ever gave glucosamine to a pet and had positive results? I would love to hear any comments - positive or negative, from you all.

Thanks for reading and have a safe Labor Day weekend!

TJ
P.S. - The rooster's name is "Rudy"...

 

[GloriaH]

I give my horses and dogs Glucosamine Chondroitin every day and it works wonders. The brand I use is made by AniMed. Natural AniFlex Complete. You can get 16 ounces for about $20. It is easier than pills as it is a powder. It has all sorts of vitamins and minerals in it. I wouldn't be afraid to try it. It probably wouldn't take much so 16 ounces would last a long time. Have you thought about warm compresses to see if that helps. I hope it helps Rudy.

 

[dlhunicorn]

can you please give details as to what is wrong with your lil roo and why you think glucosamine would "help" him...if it is a viral form of arthritis that birds get... then he would need other meds

 

[rooster-red]

I used to give it to my dog the last 3 years of his life, it seemed to help his arthritic hips.

I take it everday, it helps me alot.

 

[chickenfeathers]

I take it every day.

No more knee pain!

Don't know about it helping Rudy though.

 

[dlhunicorn]

http://www.asasea.com/po15_95.html
(excerpt)
"....Leg Weakness
Disorders involving leg weakness are a persistent problem in commercial poultry operations around the world. Leg problems in poultry are associated with many causes including nutrition, genetics, virus infection, environment. The problem is widespread with 2 to 6% of all commercial chickens displaying some sort of problem. Descriptions of the more common problems are given:
Viral arthritis
Also called tenosynovitis. Reovirus infection is considered the main cause of the problem with secondary Staphylococcus aureus infection. The disease is not nutritional in origin but may occur along with malabsorption syndrome. The condition results in severe lameness that reduces ability of the bird to move causing malnutrition and stunting. Swelling of the shanks and hock can be observed as early as 10 days but usually develop at 4 to 6 weeks. The swollen area may be filled with clear or bloody fluid or may be hardened and fibrous.

Femoral head necrosis
This condition is also called "brittle bone disease". It appears to be related to reovirus and adenovirus as these are often isolated from affected flocks. Fusarium contamination of ingredients is often observed. Femoral head necrosis is characterized by a severe weakening or disintegration of the head of the femur such that upon necropsy the bone end is easily broken off between the fingers.

Synovitis
This is caused by infection with Mycoplasma synoviae. The major signs are lameness and swelling of the hock joints with cream colored fluid. Eradication has been successful through blood testing of the breeders. Egg transmission can be reduced by dipping of eggs with antibiotic.

Osteomyelitis
This condition is caused by bacterial infection. S. aureus is the major cause with secondary involvement of E. coli and Pasteurella multocida. Invasion of bacteria after toe trimming or by cuts and scratches produce toxins that prevent cartilage formation. Birds often have a hopping gait and the affected area is swollen and warm to the touch. Biosecurity, good hygiene and treatment with antibiotics will reduce problems if caught in the early stages.

Tibial dyschondroplasia
This abnormality occurs primarily at the growth ends of the tibia where a large amount or "plug" of unvascularized cartilage accumulates. TD does not appear to be related to virus infection. Faster growing flocks on a high plane of nutrition are often affected. Acid-base imbalance, high levels of salt, low calcium and excess nitrogen increase severity of TD (Waldroup, 1986). Fusarium mycotoxins such as fusarochromanone and contamination of corn with Fusarium moniliforme increases the incidence of TD (Cook, 1987). Aflatoxin reduces vitamin D absorption and liver damage prevents conversion to the active 25-OH form of vitamin D3. Experimentally, 1, 25- OH vitamin D3 has ben found to prevent TD. Vitamin C has also been found useful. Although TD occurs in other bones, it is most common at the proximal end of the tibia because of high stress at this point. A higher incidence of breast blisters is usually observed in birds with this condition probably because they are spending more time off their feet.

Rickets
A direct result of vitamin D deficiency and low or imbalanced calcium or phosphorus nutrition. Bones are decalcified and weakened causing bowing of the legs and other problems. The growth plate is increased in width and birds appear sluggish and are reluctant to walk. The bones and beaks are soft and rubbery. Mycotoxins are often involved.

Perosis
Also called chondrodystrophy or "slipped tendon". Symptoms include swelling of the hock joint, shortening of the leg bone and gastrocnemius tendon slippage off the condyle. This problem is mostly genetic but may be induced experimentally in diets deficient in one or more of the following nutrients: choline, manganese, zinc, copper, niacin, biotin, pyridoxine, vitamin E, vitamin B12, calcium and phosphorus.

Twisted leg
This very common ailment in broilers is often confused with other problems. One or both legs may be involved. The legs may be bent inward or outward. Litter quality and heat stress seem to play a role. Manganese deficiency worsens the condition whereas high doses of pyridoxine improve the condition (Waldroup, 1986). Dietary tannin from rapeseed meal and high tannin sorghum as well as the high sulfur content in rapeseed and Canola meal interfere with calcium metabolism and increase incidence of this condition (Summers, 1993).


Strategies for reducing incidence of leg problems:
Biosecurity and disease control to eradicate mycoplasma and reduce the indicence of reovirus.

Monitor and reduce contamination of grains, groundnut meal and corn gluten meal with aflatoxin and Fusarium mold.

Calcium and phosphorus sources should be highly bioavailable. Avoid dolomitic limestone containing more than 3% magnesium as this impairs calcium utilization. Phosphate sources should contain less than 0.25% fluoride and defluorinated rock phosphate should contain between 4 to 6% sodium to ensure solubility of phosphorus. Maintain a 2:1 ratio of calcium to available phosphorus for broilers and pullets and 12:1 ratio for layers.

Ensure adequate available levels of all vitamins and trace minerals. Additional vitamin E (up to 150 ppm), biotin (up to 60 ppm) and supplementation with vitamin C (125 ppm) may be useful.

Avoid excess sodium (above 0.30%) and chloride (above 0.40%) in feed.

Avoid water with sodium above 500 ppm, chloride above 500 ppm, nitrogen (as NO3) above 50 ppm and sulfur (as sulfate) above 1000 ppm (Leeson and Summers, 1991).

Monitor sulfur level in feed. This can be a problem when using high levels of rapeseed and/or Canola meals. Total feed sulfur should be less than 0.5%.

Avoid high levels of tannins. Monitor the use of ingredients such as high tannin sorghum, sunflower meal, Canola and rapeseed meals.

Reduce nutrient density in feed to slow growth when persistent problems occur. Avoid amino acid imbalances and excess protein. .."

 

[TJ]

...Thank you all for responding. I'm not sure what it is dlhunicorn, reading your information seems as if it could be a number of those. The vet said that it wasn't swollen due to an infection where it could be lanced/opened to relieve pressure. He said it was a bone/joint issue. If I press on his legs it is as if his legs are rock hard, there is no softness to them.

I've taken photos of Rudy the Rooster so you all can see what I am talking about. He is just an absolute sweetheart.

Again, thank you for your information.

TJ

 

[silkiechicken]

Poor thing! I hope you figure it out.

 

[Pine Grove]

Typical gout, What are you feeding?

 

[TJ]

Quote:
Hi, thanks for responding... What do you mean by typical gout? And what is gout? I've heard of people getting it but never understood what it was. And I'm feeding regular laying poultry feed from the local feed store.

Any suggestions are greatly appreciated!

Thank you,

TJ