First and foremost I’m not a vet and I can’t really say what’s going on, especially only looking at photos but even in person I can’t really say what going on, a vet can diagnose better than I can, but looking at the wide leg stance with the angel wing it could be rickets.
“Rickets in poultry is caused mainly by deficiencies of either vitamin D3, calcium, phosphorus, or calcium-phosphorus imbalance. Rickets in poultry occurs frequently by using old feed, in which vitamin D3 has been destroyed by rancidity.”
From
https://tvmdl.tamu.edu/2020/03/30/rickets-in-backyard-chickens/#:~:text=Misshapen sternum and bones with wider and disorganized,which vitamin D3 has been destroyed by rancidity.
Rickets occurs most commonly in young meat birds; the main characteristic is inadequate bone mineralization. Calcium deficiency at the cellular level is the main cause, although feeding a diet deficient or imbalanced in calcium, phosphorus, or vitamin D3 can also induce this problem. Young broilers and turkey poults can exhibit lameness at ~10–14 days of age. Their bones are rubbery, and the rib cage is flattened and beaded at the attachment of the vertebrae. Rachitic birds exhibit a disorganized cartilage matrix, with an irregular vascular penetration. There is an indication of impaired metabolism of collagen precursors such as hyaluronic acid and desmosine. Rickets is not caused by a failure in the initiation of bone mineralization but rather by impairment of the early maturation of this process. There is often an enlargement of the ends of the long bones, with a widening of the epiphyseal plate. A determination of whether rickets is due to deficiencies of calcium, phosphorus, or vitamin D3, or to an excess of calcium (which induces a phosphorus deficiency) may require analysis of blood phosphorus levels and investigation of parathyroid activity.
From
https://www.merckvetmanual.com/poultry/nutrition-and-management-poultry/mineral-deficiencies-in-poultry?query=Rickets in poultry
In most field cases of rickets, a deficiency of vitamin D3 is suspected. This can be due to simple dietary deficiency, inadequate potency of the D3 supplement, or other factors that reduce the absorption of vitamin D3. Rickets can best be prevented by providing adequate levels and potency of vitamin D3supplements, and by ensuring that the diet is formulated to ensure optimal utilization of all fat-soluble compounds. Young birds have limited ability to digest saturated fats, and these undigested compounds can complex with calcium to form insoluble soaps, leading to an induced deficiency of calcium. Again, this situation cannot be diagnosed through diet assay for calcium but rather through excreta assay of this mineral. Diets must also provide a correct balance of calcium to available phosphorus. For this reason, ingredients notoriously variable in their content of these minerals, such as animal proteins, should be used with extra caution. In recent years, the use of 25(OH)D3 has become very popular as a partial replacement for vitamin D3, with reports of greatly reduced incidence of rickets, especially in poults. This metabolite is similar to that naturally produced in the liver of birds in the first step of conversion of vitamin D3 to 1,25(OH)2D3, the active form of the vitamin. The commercial form of 25(OH)D3 is therefore especially useful if normal liver metabolism is compromised in any way, such as occurs with mycotoxins or other “natural” toxins in the feed that potentially impair liver metabolism.”
Now it may not necesarily be rickets, a vet can better diagnose the problem.
Was trying to do what I could myself before things got worse.I posted a picture just taken of him. Any thoughts or advice?
