I wasn't sure what was happening with my one hen at first. I knew she had a mass inside her abdomen. But I wasn't sure what the mass was instill today. Today I decided to brave it out and find out what was going on inside her. I got my things together to do the surgery, sterilized things, and made a two inch cut below her vent ( not her actual vent ). I got inside and found the mass without searching. What I found was a mass of egg yolks about the size of a baseball. I removed the mass and stitched her up. She is now resting in a pet taxie. I have put antibiotics in her water. I will post pics soon. I was just wondering if anyone else has ever tried this?
Nope ... have performed repairs, and even extracted bullets in emergencies, but have never entered the abdominal cavity of any living creature. For certain, you can, and you should, provide this bird w/ an aspirin solution, by dissolving five 5-grain (324mg) aspirins w/in a gallon of water -- this can be safely offered free-choice to poultry throughout any periods of illness or stress.
But, indeed, I have personally been in the position your chicken was in, as I've had a surgeon enter my *own* abdominal cavity, while wide awake and fully alert, and able to watch him most skillfully save my life. However, I had the benefit of local anesthesia, and his words to walk me through (although I did almost grasp his hands w/ my own, as his scalpel plunged through the second layer of my abdominal muscles ~'-)
I've been stitched numerous times, often w/ the needle in my own hand, which is mildly uncomfortable. I've set my own bones in the field, which hurts like ... well ... a lot. And, honestly, there's been pain I wouldn't have chosen to live through, had I been given any choice, despite my ability to disassociate quite well, which is an ability most humans no longer require. And, if I were this chicken, and able to choose? I'da asked for a quick end, by your best aim.
What folks can debate, once they have a better understanding of God's design, is whether the pain endured by the chicken was severe enough to outweigh the potential benefits of the procedure you've performed, which is a decision I'd defer to you, 'cause it's
your bird.
I offer the following clinical evidence as to a chicken's response to stimuli. >
> peck here << for the full report ~'-)
A wide variety of repeated stressful or painful stimuli
can induce temporary reductions in responsiveness to
noxious stimuli (called stress-induced analgesia). Stress
from social isolation can also alter pain perception in
species that normally live in groups. The two most common
indications of social separation in domestic chicks
are distress vocalizations and stress-induced analgesia.37
Birds experiencing less stress (i.e., those held and tested
in familiar large pens) showed significantly less pain-coping
behavior compared with birds tested in novel pens.36,37
Stress-induced analgesia caused by social separation is
poorly understood. In mammals, two theories describe
the role of endogenous opioids in social separation. One
theory (social separation–opioid stimulation) suggests
that opioid system activity is stimulated by stressful experiences
such as social separation, whereas another theory
(social separation–opioid withdrawal) suggests the opposite—
social isolation places an animal in a state similar to
opioid withdrawal. Birds that become separated from
conspecifics elicit distress vocalizations in an attempt to
reestablish social contact. This isolation causes a state of
endogenous opioid withdrawal, leading to disinhibition
of distress vocalizations, whereas the presence of social
companions stimulates the release of endogenous opioids,
inhibiting vocalizations.38 Opioid agonists tend to
decrease distress vocalizations, whereas opioid antagonists
increase separation-induced distress vocalization. However,
when morphine was administered to isolated chicks,
there was no change in response to thermal nociception.
Adrenergic, cholinergic, dopaminergic, GABAergic, and
serotoninergic manipulations produce only modest effects
on distress vocalization. These results suggest that some
separation-stress behaviors are mediated by opioid systems
(i.e., distress vocalization), whereas others are mediated
by nonopioid systems (i.e., stress-induced analgesia
and hyperthermia).
Benzodiazepine agonists can modulate stress in many
animal models and thus influence the results of nociceptive
tests while having no effect on nociception itself. As
with morphine, the benzodiazepine agonist chlordiazepoxide
reverses distress vocalizations in chick social
separation, but unlike morphine, chlordiazepoxide also
reverses stress-induced analgesia. It appears that benzodiazepine
is less behavior-specific than morphine in
modulating separation-stress behaviors in chicks.38
Although stress may produce analgesia in some situations,
stress alone should not be considered necessary or
sufficient to induce analgesia.
Pain associated with trauma and disease can be
chronic and often involves inflammation. Behavioral
responses to pain are complex and can be influenced by
changes in an animal’s attention (level of awareness). In
chickens, changes in attention can significantly suppress
pain and reduce lameness during an experimentally
induced chronic pain stimulus (i.e., injection of sodium
urate into the joint, which mimics articular gout).
Hypoalgesia can be produced by diverting attention in
situations designed to increase feeding motivation or
motivation to explore. Complete analgesia or marked
hypoalgesia was observed in birds deprived of food for
16 hours and then given access to food following
sodium urate injection. This could be completely
reversed by naloxone, suggesting that the analgesia may
be opioid-mediated. When introduced to novel surroundings,
birds behaved as normal; alert birds and
attentional mechanisms are preoccupied with exploring
a new physical and/or social environment.
Distraction and attention-focusing strategies have
been used to help humans cope with chronic low-level
pain. Coping is based on the cognitive action of switching
attention; thus when patients were fully engaged in
a task, they did not process pain at the same time. The
absence of pain-related behavior does not necessarily
indicate an absence of pain. Expression of pain in birds,
similar to that in humans, can be altered by the motivational
state of the individual, but the analgesia experienced
is likely only temporary.
Although the precise mechanism is unknown, there is
evidence that the peripheral nervous system plays a significant
role in inflammation. Some evidence suggests
that changes in attention resulting in reduced pain may
also directly influence inflammation, leaving only the
general tissue reaction. Studies on the neural activity in
the medullary dorsal horn of monkeys suggest that
attention-dependent changes in sensory discrimination
and affective components of pain are mediated at the
early stages of sensory processing. If similar changes
occur while processing nociceptive information at the
spinal level, the activity of the peripheral nervous system
may also be affected. Reduced pain perception and
inflammation were demonstrated following attentional
changes in chickens. However, more research is necessary
before clear conclusions can be drawn.
1. Gottschaldt K-M, Fruhstorfer H, Schmidt W: Thermosensitivity and its
possible fine-structural basis in mechanoreceptors in the beak skin of geese. J
Comp Neurol 205:219–245, 1982.
2. Necker R, Reiner B: Temperature-sensitive mechanoreceptors, thermoreceptors
thermoreceptors
and heat nociceptors in the feathered skin of pigeons. J Comp Physiol
135:201–207, 1980.
3. Gentle MJ, Hunter LN: Physiological and behavioural responses associated
with feather removal in Gallus gallus var domesticus. Res Vet Sci 50:95–101, 1990.
4. Hocking PM, Gentle MJ, Bernard R, Dunn LN: Evaluation of a protocol for
determining the effectiveness of pretreatment with local analgesics for reducing
experimentally induced articular pain in domestic fowl. Res Vet Sci
63:263–267, 1997.
5. Hughes RA, Bowes M, Sufka KJ: Morphine hyperalgesic effects on developmental
changes in thermal nociception and respiration in domestic fowl
(Gallus gallus). Pharmacol Biochem Behav 42:535–539, 1992.
6. Curro TG: Evaluation of the isoflurane-sparing effects of butorphanol and
flunixin in psittaciformes. Proc Assoc Avian Vet 17–19, 1994.
7. Paul-Murphy JR, Brunson DB, Miletic V: Analgesic effects of butorphanol
and buprenorphine in conscious African grey parrots (Psittacus erithacus erithacus
and Psittacus erithacus timneh). Am J Vet Res 60(10):1218–1221, 1999.
8. Reiner A, Davis BM, Brecha N: The distribution of enkephalinlike
immunoreactivity in the telencephalon of the adult and developing domestic
chicken. J Comp Neurol 228:245–262, 1984.
9. Curro TG, Brunson DB, Paul-Murphy J: Determination of the ED50 of
isoflurane and evaluation of the isoflurane-sparing effect of butorphanol in
cockatoos (Cacatua spp). Vet Anesth 23:429–433, 1994.
10. Danbury TC, Chambers JP, Weeks CA: Self-selection of analgesic drugs by
broiler chickens. Anim Choices 20:126–127, 1997.
11. Livingston A: Physiological basis for pain perception in animals. J Vet Anaesth
21:73–77, 1994.
12. Gentle MJ: Pain in birds. Anim Welfare 1:235–247, 1992.
13. Chapman CR, Casey KL, Dubner R: Pain measurement: An overview. Pain
22:1–31, 1985.
14. Gentle M: The acute effects of amputation on peripheral trigeminal afferents
in Gallus gallus var domesticus. Pain 46:97–103, 1991.
15. Holloway JA, Trought CO,Wright LE: Cutaneous receptive field characteristics
of primary afferents and dorsal horn cells in the avian (Gallus domesticus).
Exp Neurol 68:477–488, 1980.
16. Breward J, Gentle MJ: Neuroma formation and abnormal afferent nerve discharges
after partial beak amputation (beak trimming) in poultry. Experientia
41:1132–1134, 1985.
17. Nicol GD, Klingberg DK, Vasko MR: Prostaglandin E2 increases in calcium
conductance and stimulates release of substance P in avian sensory neurons. J
Neurosci 12(5):1917–1927, 1992.
18. Zhai S-Y, Atsumi S: Large dorsal horn neurons which receive inputs from
numerous substance P–like immunoreactive axon terminals in the laminae I
and II of the chicken spinal cord. Neurosci Res 28:147–154, 1997.
19. Wan Q, Pang SF: Segmental, coronal and subcellular distribution of 2-
[125I]iodomelatonin binding sites in the chicken spinal cord. Neurosci Lett
180:253–256, 1994.
20. Fernandez-Lopez A, Revilla V, Candelas MA: A comparative study of
alpha2- and beta-adrenoceptor distribution in pigeon and chick brain. Eur J
Neurosci 9(5):871–883, 1997.
21. Danbury TC, Hudson AL,Waterman-Pearson AE: Saturation binding of μ, d,
and k opioid ligands in chicken brains. Arch Pharm 358(suppl 35)(1):105, 1998.
22. Hendrickson CM, Lin S: Opiate receptors in highly purified neuronal cell
populations isolated in bulk form embryonic chick brain. Neuropharmacology
13:731–739, 1980.
23. Hocking PM, Bernard R, Maxwell MH: Assessment of pain during locomotion
and the welfare of adult male turkeys with destructive cartilage loss of
the hip joint. Brit Poult Sci 40:30–34, 1999.
24. Gentle MJ, Tilston VL: Reduction in peripheral inflammation by changes in
attention. Physiol Behav 66(2):289–292, 1999.
:: edited to add the link:
http://mobile.vetlearn.com/Media/PublicationsArticle/PV_27_02_98.pdf
/edit ::