@Chooks man I just noticed one of my LA pullets I am growing out has some odd webbing between her toes. There is webbing between the middle toe and outer toe on both feet. This is a serious defect and disqualification among show birds but how hard of an impact is it genetically in regard to their use as breeding stock? Is it something I should cull now? I don't want to use chickens with genetic defects for breeding to carry forward. What are your thoughts on this? I drew a picture of what I'm talking about on my laptop because after I saw the pullet and went inside to get my phone, I couldn't find her again when I came back out. I will look for her again when I feed and grab a pic. She is the only bird I have produced that has shown this defect.
View attachment 1204691
Here is some information I found on BYC posted on another forum.
"Syndactyly is a condition in which the third and fourth toes are fused, usually by continuous webbing between the toes (Figure 6.6). The extent of webbing varies distally and between toes. In extreme cases the toes are tightly tied together, and the nail of the fourth toe may be bent out of its median position making walking difficult. The condition is usually expressed in both feet. Syndactyly has been associated with both ptilopody and brachydactyly by several workers (Davenport, 1909; Danforth, 1919a, 1919b; Jaap, 1939). They proposed that the same gene was responsible for all three of these conditions and that modifying genes and genetic background determine the phenotype of each individual. Davenport's (1909) data showed syndactyly to be dominant. This would be expected if one of the dominant shank feathering genes were responsible for it. Danforth (1929) suggested that both of the dominant shank feathering genes were able to produce syndactyly. A relationship between these two traits was further strengthened with the appearance of the single gene multitrait mutant psp reported by McGibbon and Shackelford (192) and Smyth (1981) in which syndactyly and ptilopody were shown to be caused by the same recessive gene.
There probably is more than one genetic type of syndactyly. Two studies have been made of this trait in clean-legged stock, where ptilopody genes could not be involved. One study used White Plymouth Rocks (Warren, 1950) and the other one involved junglefowl stock (Hollander and Brumbaugh, 1969). In both cases, results from crosses were difficult to interpret. Warren (1950) found that the normal parent had a major influence on F1 progeny; he got only 1.8 percent syndactylism in one group of F1 crosses but 41 percent in another, syndactyl x syndactyl crosses produced 62 percent affected progeny, and there were only 11.9 percent affected in the F2 generation. The comparable results from Hollander and Brumbaugh (1969) were zero, 40 and one percent affected birds. Although Warren (1950) had somewhat more affected individuals, these two groups may well have been working with the same genetic complex. Hollander and Brumbaugh (1969) proposed complementary gene action of a dominant and a recessive factor along with variable penetrance and expressivity. Warren (1950) did not suggest a mode of inheritance, but said that there was no evidence of sex-linkage."
pp. 201-202, Poultry Breeding and Genetics -- Roy D. Crawford, Elsevier, 1990.